Rapamycin Preserves Neuron Health in Alzheimer's

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Physionic video on Rapamycin, published on November 20, 2024

Quotes From Source

“The membrane potential is compromised in Alzheimer's disease...”

Story of claim

Rapamycin may help preserve neuron health by recovering mitochondrial membrane potential and reducing pro-apoptotic proteins like Bax in Alzheimer's models.

  • Goal: To enhance mitochondrial function and reduce cell death proteins in Alzheimer's disease.
  • Proof: Recovery of mitochondrial potential and reduced pro-apoptotic proteins indicate neuron preservation effects.
  • Nuances:
    • Involves Parkin enzyme
    • Potential reduction in toxic proteins like tau
  • Impact on Life: May sustain neuronal function, potentially improving mental health and slowing Alzheimer's progression.

Investments

  • Price: Costs depend on research and treatment specifics
  • Time: Effects on neuronal health may take time to observe
  • Effort: Requires consistent monitoring and treatment adjustments

Risks

Potential for adverse effects if not carefully managed, especially concerning mitochondrial health.

Alternatives

  • Other mitochondrial-targeted therapies
  • Neuroprotective lifestyle changes

Get Started 🚀

  • Engage with medical professionals on Rapamycin's use
  • Follow new research in neurodegenerative treatments
  • Consider joining clinical studies for emerging therapies

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Reference Video